Polymorphous light eruption ... |
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The papular type is the most common form. Small papules are densely aggregated.
The back of the hands is the most common site. |
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DESCRIPTION
Idiopathic, recurrent photodermatitis that comes on acutely, usually in spring with first sun exposure.
HISTORY
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Commonly referred to as sun poisoning or sun allergy.
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Occurs in all races, at any age, but most common in young women, in whom the incidence is as high as 10% in the population.
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Occurs in northern climates where sun intensity increases in spring, and during winter vacations while visiting southern, sunny resorts.
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Symptoms may include pruritus, malaise, chills, headache, nausea.
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Usually appears with first one or two exposures to sunlight in spring. Ultraviolet A is trigger in most cases, although the amount of exposure needed to trigger a flare varies.
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Hereditary polymorphous light eruption occurs in Inuit and Native American people.
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Autosomal dominant transmission with incomplete penetrance and variable expressivity.
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PHYSICAL FINDINGS
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Initial symptoms are burning, itching, and erythema on exposed skin such as upper chest, backs of hands, extensor aspects of forearms, and lower legs. Lesions appear 2 h to 5 days after sun exposure. Tends to spare the face (except in hereditary form).
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Several clinical types. Papular type: most common; small red dermal papules, disseminated or densely aggregated, on patchy erythema. Plaque type: second most common; superficial or urticarial plaques. Papulovesicular type-, least common; begins with urticarial plaques from which groups of vesicles arise. Affected patients develop the same clinical type of polymorphous light eruption each year.
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Lesions persist for 7-10 days.
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Light sensitivity decreases with repeated sun exposure into the summer and recurs the next spring. Most patients have exacerbations each summer for many years.
TREATMENT
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Minimize sun exposure, especially between 10 a.m. and 2 p.m.
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Broad-spectrum, ultraviolet A-blocking sunscreens, especially those containing avobenzone, Mexoryl, titanium dioxide, and zinc oxide.
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Using group II—V topical steroids for 3-14 days relieves itch and fades lesions.
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Oral steroids (tapered gradually over 2 weeks) useful in severe cases.
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Hydroxychloroquine 400 mg/day for first month and 200 mg/ day thereafter for difficult cases.
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Controlled gradual exposure to sunlight or to ultraviolet B or ultraviolet A can harden skin and increase tolerance.
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| Courtesy by : Thomas P. Habif, James L. Campbell Jr, M. Shane Chapman, James G. H. Dinulos, Kathryn A. Zug |
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